Harman Haini Group

The development of oral mucositis follows a complex biological process triggered by cancer therapies. Chemotherapy and radiation damage rapidly dividing epithelial cells, leading to inflammation, tissue breakdown, and ulcer formation. This process involves oxidative stress, cytokine release, and immune system activation.

As the mucosal barrier weakens, patients become vulnerable to infections and severe pain. Understanding the underlying mechanisms helps clinicians design targeted prevention and treatment strategies. Research into molecular pathways continues to improve therapeutic approaches.

FAQ: Q: What causes tissue damage in oral mucositis? A: Cancer therapies damage fast-growing oral epithelial cells.